Category: Enterococcus

Lines on lines on lines: CLABSI, CRBSI, and Line Infections

Central lines are cool, for the most part. Except for when they’re infected. The pathophysiology behind these is explained in this diagram (1): Microorganisms gain access to the extraluminal or intraluminal surface of the device. Both skin and hub may be the important entry point of organisms, with the development of biofilm contributing to their

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Daptomycin vs Linezolid for Vancomycin-Resistant Enterococcus

I’ve previously discussed how much more difficult Enterococcal infections are to treat when compared to other bacteria such as streptococci. This stems from the fact these bacteria have tend to be more resistant to beta-lactam antibiotics, with synergy being the name of the game for serious infections with these bacteria (in general, ampicillin/ceftriaxone). This is

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Enterococcal bacteremia and the risk of infective endocarditis – No Go

Enterococcal infections and bacteremia incidence have been increasing over the past decades, with enterococcal endocarditis representing 10% of all cases of infective endocarditis (1). This makes it the third most common organism implicated in IE, after Staphylococcus and streptococcus, and it is more prevalent in patients who are not drug abusers. Enterococcal bacteremia by itself

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